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rabbit polyclonal antibody against sam68 (Danaher Inc)

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Danaher Inc rabbit polyclonal antibody against sam68
Rabbit Polyclonal Antibody Against Sam68, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 99/100, based on 20243 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+antibody+against+sam68/Rabbit+Polyclonal+Anti-JAK2+(phospho+Y1007)+antibody/pmc06600183-379-0-8
Average 99 stars, based on 20243 article reviews

rabbit polyclonal antibody against sam68 - by Bioz Stars, 2026-10

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Article Title: Sam68 Promotes Hepatitis C Virus Replication by Interaction with Stem-Loop 2 of Viral 5′ Untranslated Region
Article Snippet: Rabbit polyclonal antibody against Sam68 was obtained from Abcam.

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Correlation of Sam68 expression with clinicopathological characteristics of EOC patients.

Journal: Medical Science Monitor : International Medical Journal of Experimental and Clinical Research

Article Title: Sam68 is Overexpressed in Epithelial Ovarian Cancer and Promotes Tumor Cell Proliferation

doi: 10.12659/MSM.899980

Figure Lengend Snippet: Correlation of Sam68 expression with clinicopathological characteristics of EOC patients.

Article Snippet: Western blotting was performed using rabbit polyclone antibody against human Sam68 (Santa Cruz Biotechnology, Santa Cruz, CA, USA), or rabbit polyclone antibody against β-actin antibody (Sigma-Aldrich, St. Louis, MO, USA).

Techniques: Expressing, Biomarker Discovery

Overexpression of Sam68 in ovarian cancers. ( A ) Relative mRNA level of Sam68 in the ovarian cancer specimens (n=152) with para-tumor tissues as control. ( B ) Representative Western blot analysis of Sam68 protein level in ovarian cancer and para-tumor specimens. ( C ) Relative protein level of Sam68 to β-actin in the ovarian cancer and para-tumor tissues (n=30). Paired t -test was performed to examine the difference in either mRNA or protein level of Sam68, and statistical significance was considered when p <0.05.

Journal: Medical Science Monitor : International Medical Journal of Experimental and Clinical Research

Article Title: Sam68 is Overexpressed in Epithelial Ovarian Cancer and Promotes Tumor Cell Proliferation

doi: 10.12659/MSM.899980

Figure Lengend Snippet: Overexpression of Sam68 in ovarian cancers. ( A ) Relative mRNA level of Sam68 in the ovarian cancer specimens (n=152) with para-tumor tissues as control. ( B ) Representative Western blot analysis of Sam68 protein level in ovarian cancer and para-tumor specimens. ( C ) Relative protein level of Sam68 to β-actin in the ovarian cancer and para-tumor tissues (n=30). Paired t -test was performed to examine the difference in either mRNA or protein level of Sam68, and statistical significance was considered when p <0.05.

Techniques: Over Expression, Control, Western Blot

Univariate analysis of prognostic factors in EOC patients.

Journal: Medical Science Monitor : International Medical Journal of Experimental and Clinical Research

Article Title: Sam68 is Overexpressed in Epithelial Ovarian Cancer and Promotes Tumor Cell Proliferation

doi: 10.12659/MSM.899980

Figure Lengend Snippet: Univariate analysis of prognostic factors in EOC patients.

Techniques: Biomarker Discovery, Expressing

Multivariate analysis of prognostic factors in OC patients.

Journal: Medical Science Monitor : International Medical Journal of Experimental and Clinical Research

Article Title: Sam68 is Overexpressed in Epithelial Ovarian Cancer and Promotes Tumor Cell Proliferation

doi: 10.12659/MSM.899980

Figure Lengend Snippet: Multivariate analysis of prognostic factors in OC patients.

Techniques: Expressing

pRC/CMV-mediated upregulation and siRNA-mediated knockdown of Sam68 in ovarian cancer OVCAR-3 cells ( A ) mRNA level of Sam68 in OVCAR-3 cells, which were transfected with pRC/CMV-Sam68-FLAG (Sam68 +) or pcRC/CMV (Con +, as control); ( B ) Western blot analysis of Sam68 in the blank, Sam68 +, or Con + OVCAR-3 cells; ( C ) mRNA level of Sam68 in the OVCAR-3 cells, which were transfected with siRNA-Con or siRNA-Sam68 (20 or 40 nM); ( D ) Western blot analysis of Sam68 in the siRNA-Con- or siRNA-Sam68-transfected (20 or 40 nM) OVCAR-3 cells. Data was averaged for triple independent results. ns represented no significance, ** represented p <0.01, and *** represented p <0.001.

Journal: Medical Science Monitor : International Medical Journal of Experimental and Clinical Research

Article Title: Sam68 is Overexpressed in Epithelial Ovarian Cancer and Promotes Tumor Cell Proliferation

doi: 10.12659/MSM.899980

Figure Lengend Snippet: pRC/CMV-mediated upregulation and siRNA-mediated knockdown of Sam68 in ovarian cancer OVCAR-3 cells ( A ) mRNA level of Sam68 in OVCAR-3 cells, which were transfected with pRC/CMV-Sam68-FLAG (Sam68 +) or pcRC/CMV (Con +, as control); ( B ) Western blot analysis of Sam68 in the blank, Sam68 +, or Con + OVCAR-3 cells; ( C ) mRNA level of Sam68 in the OVCAR-3 cells, which were transfected with siRNA-Con or siRNA-Sam68 (20 or 40 nM); ( D ) Western blot analysis of Sam68 in the siRNA-Con- or siRNA-Sam68-transfected (20 or 40 nM) OVCAR-3 cells. Data was averaged for triple independent results. ns represented no significance, ** represented p <0.01, and *** represented p <0.001.

Techniques: Knockdown, Transfection, Control, Western Blot

Sam68 upregulation promotes the proliferation of ovarian cancer OVCAR-3 cells. ( A ) Growth curve of the OVCAR-3 cells, in which Sam68 was upregulated (Sam68 [Up]) or not (Control [Up]), as was determined by CCK-8 assay; ( B ) Colony forming assay of the Sam68 (Up) or Control (Up) OVCAR-3 cells, which were incubated for 48 hours; ( C, D ) Colony counting ( C ) and colony size ( D ) of the colony which was formed by the Sam68 (Up) or Control (Up) OVCAR-3 cells. Experiments were repeated in quadruplicate independently. ANOVA or unpaired t -test was performed to examine the difference in the proliferation of colony formation between the Sam68 (Up) and Control (Up) OVCAR-3 cells. Data were averaged for triple independent results. Statistical significance was considered when p <0.05. * represented p <0.05 and ** represented p <0.01.

Journal: Medical Science Monitor : International Medical Journal of Experimental and Clinical Research

Article Title: Sam68 is Overexpressed in Epithelial Ovarian Cancer and Promotes Tumor Cell Proliferation

doi: 10.12659/MSM.899980

Figure Lengend Snippet: Sam68 upregulation promotes the proliferation of ovarian cancer OVCAR-3 cells. ( A ) Growth curve of the OVCAR-3 cells, in which Sam68 was upregulated (Sam68 [Up]) or not (Control [Up]), as was determined by CCK-8 assay; ( B ) Colony forming assay of the Sam68 (Up) or Control (Up) OVCAR-3 cells, which were incubated for 48 hours; ( C, D ) Colony counting ( C ) and colony size ( D ) of the colony which was formed by the Sam68 (Up) or Control (Up) OVCAR-3 cells. Experiments were repeated in quadruplicate independently. ANOVA or unpaired t -test was performed to examine the difference in the proliferation of colony formation between the Sam68 (Up) and Control (Up) OVCAR-3 cells. Data were averaged for triple independent results. Statistical significance was considered when p <0.05. * represented p <0.05 and ** represented p <0.01.

Techniques: Control, CCK-8 Assay, Incubation

Sam68 knockdown inhibits the proliferation of ovarian cancer OVCAR-3 cells. ( A ) Growth curve of the OVCAR-3 cells, in which Sam68 was knocked down (Sam68 [KD]) or not (Control [KD]), as was determined by CCK-8 assay. ( B ) Colony forming assay of the Sam68 (KD) or Control (KD) OVCAR-3 cells, which were incubated for 96 hours. ( C, D ) Colony counting ( C ) and colony size ( D ) of the colony which was formed by the Sam68 (KD) or Control (KD) OVCAR-3 cells. Experiments were repeated in quadruplicate independently. ANOVA or unpaired t -test was performed to examine the difference in the proliferation of colony formation between the Sam68 (KD) and Control (KD) OVCAR-3 cells. Data was averaged for triple independent results. Statistical significance was considered when p <0.05. ns represented no significance, * represented p <0.05, and ** represented p <0.01.

Journal: Medical Science Monitor : International Medical Journal of Experimental and Clinical Research

Article Title: Sam68 is Overexpressed in Epithelial Ovarian Cancer and Promotes Tumor Cell Proliferation

doi: 10.12659/MSM.899980

Figure Lengend Snippet: Sam68 knockdown inhibits the proliferation of ovarian cancer OVCAR-3 cells. ( A ) Growth curve of the OVCAR-3 cells, in which Sam68 was knocked down (Sam68 [KD]) or not (Control [KD]), as was determined by CCK-8 assay. ( B ) Colony forming assay of the Sam68 (KD) or Control (KD) OVCAR-3 cells, which were incubated for 96 hours. ( C, D ) Colony counting ( C ) and colony size ( D ) of the colony which was formed by the Sam68 (KD) or Control (KD) OVCAR-3 cells. Experiments were repeated in quadruplicate independently. ANOVA or unpaired t -test was performed to examine the difference in the proliferation of colony formation between the Sam68 (KD) and Control (KD) OVCAR-3 cells. Data was averaged for triple independent results. Statistical significance was considered when p <0.05. ns represented no significance, * represented p <0.05, and ** represented p <0.01.

Techniques: Knockdown, Control, CCK-8 Assay, Incubation

Immunoprecipitation of UV-crosslinked proteins. Cytoplasmic extracts from HeLa (lanes 1–4), DBT (lanes 5–8), and A59-infected DBT cells (lanes 9–12) were UV-crosslinked with 32P-labeled (−)-strand leader RNA. Different antibodies were used for immunoprecipitation, and the precipitated proteins were analyzed by SDS/PAGE, a, mAb for hnRNP A1, 4B10; b, polyclonal antibody for TFIIB; and c, polyclonal antibody for Sam68. The asterisk indicates the potential protein specific for MHV-infected cells.

Journal:

Article Title: Heterogeneous nuclear ribonucleoprotein A1 binds to the transcription-regulatory region of mouse hepatitis virus RNA

doi:

Figure Lengend Snippet: Immunoprecipitation of UV-crosslinked proteins. Cytoplasmic extracts from HeLa (lanes 1–4), DBT (lanes 5–8), and A59-infected DBT cells (lanes 9–12) were UV-crosslinked with 32P-labeled (−)-strand leader RNA. Different antibodies were used for immunoprecipitation, and the precipitated proteins were analyzed by SDS/PAGE, a, mAb for hnRNP A1, 4B10; b, polyclonal antibody for TFIIB; and c, polyclonal antibody for Sam68. The asterisk indicates the potential protein specific for MHV-infected cells.

Article Snippet: The polyclonal rabbit antibodies against Sam68 and TFIIB were purchased from Santa Cruz Biotechnology.

Techniques: Immunoprecipitation, Infection, Labeling, SDS Page

The relocalization of hnRNP A1 in MHV-infected cells. Immunofluorescent staining of MHV-infected cells (×200 magnification) at various time points postinfection. hnRNP A1 or Sam68 antibodies were used.

Journal:

Article Title: Heterogeneous nuclear ribonucleoprotein A1 binds to the transcription-regulatory region of mouse hepatitis virus RNA

doi:

Figure Lengend Snippet: The relocalization of hnRNP A1 in MHV-infected cells. Immunofluorescent staining of MHV-infected cells (×200 magnification) at various time points postinfection. hnRNP A1 or Sam68 antibodies were used.

Article Snippet: The polyclonal rabbit antibodies against Sam68 and TFIIB were purchased from Santa Cruz Biotechnology.

Techniques: Infection, Staining

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rabbit polyclonal antibody against sam68 (Danaher Inc)

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